Could be a problem with other PT's who all they want to do is make it so that THEY fix the pain, and the patient is reliant to them...keeps 'em coming back I guess...
TotalMotionRelease.com

Below is an interview of Frank Forencich of Exuberant Animal. The interview was done by Chris at Conditioning Research.
Modern society seems to be sad! I keep hearing of friends or family members who are depressed or suffering anxiety attacks. How much of this stress and dissatisfaction do you think comes from our “modern” lifestyles?
What can we do about it?
"There's been a LOT of work in the past 4-5 years about new research in foot ware and care. Guess what? feet work. All by themselves.
Guess what else? shoes stop feet from working. That's a pretty global condemnation but it's true: with a handful of exceptions, modern shoes are based on 200 year old technology (the lasts of shoe design), and for the most part are way way way too restrictive to let our feet do their thing.
It's not just four inch heels or wing tips that are the problem: it's also flip flops and horror of horrors those gorgeous high tech trainers with designs to "correct" supination or too much pronation or heel strike or whatever. And just when you realize that that's as bad as putting the foot into a cast, we find that flip flops and Birkenstocks sandles are equally horrific for other reasons: toes have to claw onto the sandle to keep them on. Despite claims that such "foot muscle work" is good for you, it really isn't. The body doesn't keep our feet in flexion (toes curled) with every step we take when we walk barefoot. Why? Our feet are one of the most jointed parts of our body (after the skull and the hands) and yet daily, what do we do? Lace up shoes to restrict those bones from doing what they were designed to do to support us: MOVE.THere's a fantastic piece in the New York Magazine from earlier this year that describes most of the latest research and why shoes suck. Recommended reading.
Lots of joints in the foot, huh?
One benefit of freeing the feet this article doesn't touch on is the relation of squished feet to the nervous system. We don't talk about the nervous system much, it's just sorta there, right? But here's the thing: the nervous system, as described by Eric Cobb, is hard wired to check only very few things. One of these, demonstrated in the startle reflex, is not fight or flight, but the very binary Threat or No Threat. "We're geared to optimize for survival, not performance," according to Cobb. Most of the nerves in our bodies designed to detect how we're moving in space are at the joints. Guess what happens in terms of that Threat/No Threat thing if our joints are squished and so not sending happy "we're free and moving" signals back to the rest of the system? Is that going to be interpretted as a Threat or a No Threat?
As Cobb demonstrates in his seminars, because we're totally connected systems, optimized for survival, if we get a message somewhere in our body that says there's a signal interruption, other parts of the body respond - and they respond immediately. In one demo, Cobb did a muscle test on a barefooted athlete to check for hamstring (back of the leg) strength. Rock solid. He then simply grabbed the athlete's foot, holding it snugly as in a laced shoe, and did the muscle test again. It was like those leg muscles got unplugged. Why?
This shut down response is part of the signaling process that says if there's something wrong somewhere, we your nervous system, don't want you exerting effort that could put you at further risk. Attend!
So above and beyond all the amazing stories about how shoes are bad for us biomechanically - because they get in the way of our own vastly superior biomechanics - they're also bad for us neurologically. Squished or non-mobile joints tell our body there's a problem. Every step we take with these immobilized joints sends that message "there's a problem; there's a problem: threat threat threat."
As most of us have experienced, if we don't attend to the quiet signals, our body has a way of sending messages out to get attention. And not necessarily at the site of the problem. Restricted feet lead to knee issues, or a hip issue or back issue, or shoulder ache or a jaw pain or maybe a wrist pain, to name a few hot spots."
This post was taken from 'IamgeekFit' blogger. It struck some cords with me as to what I've been trying to tell folks for a while now. Here's another example--for ladies who wear pointy toed, high heels, what is the first thing you do when you take your shoes off? Take a deep breath right? That's because the feet have been CONFINED.
You go geek fit dude...
It's a safe guess that somewhere at Merck today someone is going through the meeting minutes of the day that the hair-brained scheme for the Australasian Journal of Bone and Joint Medicine was launched, and that everyone who was in the room is now going to be fired.
The Scientist has reported that, yes, it's true, Merck cooked up a phony, but real sounding, peer reviewed journal and published favorably looking data for its products in them. Merck paid Elsevier to publish such a tome, which neither appears in MEDLINE or has a website, according to The Scientist.
What's wrong with this is so obvious it doesn't have to be argued for. What's sad is that I'm sure many a primary care physician was given literature from Merck that said, "As published in Australasian Journal of Bone and Joint Medicine, Fosamax outperforms all other medications...." Said doctor, or even the average researcher wouldn't know that the journal is bogus. In fact, knowing that the journal is published by Elsevier gives it credibility!
These kinds of endeavors are not possible without help. One of The Scientist's most notable finds is a Australian rheumatologist named Peter Brooks who served on the "honorary advisory board" of this "journal". His take: "I don't think it's fair to say it was totally a marketing journal", apparently on the grounds that it had excerpts from peer-reviewed papers. However, in his entire time on the board he never received a single paper for peer-review, but because he apparently knew the journal did not receive original submissions of research. This didn't seem to bother him one bit. Such "throwaways" of non-peer reviewed publications and semi-marketing materials are commonplace in medicine. But wouldn't that seem odd for an academic journal? Apparently not. Moreover, Peter Brooks had a pretty lax sense of academic ethics any way: he admitted to having his name put on a "advertorial" for pharma within the last ten years, says The Scientist. An "advertorial"? Again, language unfamiliar to us in the academic publishing world, but apparently quite familiar to the pharmaceutical publishing scene.
It is this attitude within companies like Merck and among doctors that allows scandals precisely like this to happen. While the scandals with Merck and Vioxx are particularly egregious, we know they are not isolated incidents. This one is just particularly so. If physicians would not lend their names or pens to these efforts, and publishers would not offer their presses, these publications could not exist. What doctors would have as available data would be peer-reviewed research and what pharmaceutical companies produce from their marketing departments--actual advertisements.
Summer Johnson, PhD
So, who are the drug companies working for? YOUR health, or THEIR wallets? You decide.
This is my first blog. What I’ll try to do is share thoughts that might be useful in furthering my goal of getting people more active, thus healthier and happier. I want to start with the big picture as I see it. Most Americans are very inactive, and the rest of the world is trying to catch up with us. Based on accelerometer monitoring in the NHANES study, fewer than 10% of teenagers and 5% of adults meet physical activity guidelines. The real number is probably higher because accelerometers miss some activities, but I’m sure it is closer to the truth than surveys. If 95% of adults smoked, we would consider it a health crisis, but that’s about where we are with physical inactivity. We should consider it a crisis.
The epidemics of obesity and type 2 diabetes in adults and children are indications that low activity levels, along with disastrous eating habits, are the biggest health challenges of our time. I suggest you check out www.3four50.org. The basic idea is that 3 behaviors—smoking, inactivity, poor diet—are the main causes of four diseases—heart disease, cancers, lung disease, diabetes—that account for 50% of deaths worldwide. Physical inactivity is one of the big three, so we need to be serious about improving the situation.
I want to end on a positive note. With support from the Centers for Disease Control and Prevention, as well as several health organizations, a National Plan for Physical Activity is being developed. Having a plan does not ensure we will be successful, but not having a plan is a good way to guarantee continued failure. There are opportunities to have input into the development of The Plan, and it is essential that every person who believes physical activity is essential for health is directly involved in the implementation of plan. As a first step, visit www.physicalactivityplan.org.
To be proactive, we need to start viewing body weight activities with our youth as “skills” instead of “exercises.” In the good old days, kids developed many physical skills on their own through general daily activity. When an instructor would have them do a push-up, the summation of their daily tasks would allow for the strength and stability to do so. Pushing their body weight away from the ground was a demonstration of their coordination and strength. They didn’t really need to practice it much because of their active, adaptive neural systems. In those days, you could just throw “exercises” out at the kids and they could do them pretty well with some basic coaching. It doesn’t work that way anymore.
With the inactivity problem, there is hardly any strength and coordination to “showcase” in an exercise. A push-up has to be a learned skill. It has to be adapted, progressed, and practiced. Even general movement tasks like bear crawls, crab walks, and skips have to be acquired as a skill."
http://www.elitefts.com/documents/too_fat.htmWell said. Never too early to start...
Finally, the Chart You’ve Been Waiting For
Ok, now you’ve hopefully got a better idea of which diet approach may be the most ideal for you. To make it a little more clear, I’m going to try to summarize all of the above information into a chart so you can see how the different variables interact.
Diet | Activity Level | Insulin Sensitivity | Carb Choices | Carb Addict | Stubborn Fat |
High-carb/low-fat | High | High | Low GI | No | NO |
Mod carb/mod-fat | Medium | Low-moderate | Medium GI | Maybe | Yes/Maybe |
Standard Keto | Low | Low | N/A | Yes | Yes |
Targeted Keto | High | Low | N/A | Yes | Yes |
Cyclical Keto | High | Low | N/A | Yes | Yes |
The stress response—the body’s hormonal reaction to danger, uncertainty or change—evolved to help us survive, and if we learn how to keep it from overrunning our lives, it still can. In the short term, it can energize us, “revving up our systems to handle what we have to handle,” says Judith Orloff, a psychiatrist at UCLA. In the long term, stress can motivate us to do better at jobs we care about. A little of it can prepare us for a lot later on, making us more resilient.
Herein lies a problem. A lot of us tend to flip the stress-hormone switch to “on” and leave it there. At some point, the neurons get tired of being primed, and positive effects become negative ones. The result is the same decline in health that Selye’s rats suffered. Neurons shrivel and stop communicating with each other, and brain tissue shrinks in the hippocampus and prefrontal cortex, which play roles in learning, memory and rational thought. “Acutely, stress helps us remember some things better,” says neuroendocrinologist Bruce McEwen of Rockefeller University. “Chronically, it makes us worse at remembering other things, and it impairs our mental flexibility.”
So there you have it, we thrive on short bits of stress. We work our best when under a little pressure. We are healthier when we stress our body into adapting and making it more resistant for the future. It’s our lack of control of the stress over ourselves (mostly mentally) that can cause the big danger in our lives. Times are tough, nothing is easy…but it shouldn’t have to be. Most of us are not going to move to a monastery on a mountain-top to meditate full time, so we better learn how to deal with what we have going on. You don’t have to run away from things, just learn how to handle and control it. Keep your head, realize you have full control to take action in life and then just take it one moment/action at a time. Learn to use the stressors to make you stronger, and you may just live longer because of it (while others who are not able to control them, take themselves into a negative downward spiral)."Try making a commitment to getting into the meditation posture at least once a day. You don't have to sit for any particular length of time, just get on the cushion. A lot of times, the hardest part is getting there. Once you're sitting down, you think, 'I might as well sit for a few minutes,' and more often than not, you're getting full sessions in." –Insight Meditation Society co-founder Joseph Goldstein
"Try making a commitment to pick up your kettlebell at least once a day. You don't have to swing it for a particular length of time, just pick it up. Alot of times, the hardest part is getting there. Once you've picked it up, you might think, "I might as well swing for a few minutes" and more often than not, you're getting full sessions in" - Simple StrengthBack to basics once more. Turn up. That is all that is required. The rest takes care of itself.
Is this a coincidence, this similarity between the ancient home of human beings and their modern day preference? Animals of all kinds possess an inborn habitat selection on which their survival depends. It would seem strange if humans were an exception, or if humans brief existence in agricultural and urban surroundings had erased the propensity of our genes. Consider a New York multimillionaire who, provided by wealth with a free choice of habitation, selects a penthouse overlooking Central Park, in sight of the lake if possible, and rims the terrace with potted shrubs. Is the habitat we choose written in our genes?"
So, I ask you, where do you want to live? When finished answering that question, now ask yourself with this kind of DNA link to early man, do we still not believe that we are supposed to MOVE? Do we still not believe that we are becoming LAZIER each year? Do we still not believe that our DNA tells us that if we don't move that we will continue to become an unhealthy, faltering society?
I'll live here...
If you "feel the burn," you need to bulk up your mitochondria
By Robert Sanders, Media Relations | 19 April 2006
BERKELEY – In the lore of marathoners and extreme athletes, lactic acid is poison, a waste product that builds up in the muscles and leads to muscle fatigue, reduced performance and pain.
Some 30 years of research at the University of California, Berkeley, however, tells a different story: Lactic acid can be your friend.
Coaches and athletes don't realize it, says exercise physiologist George Brooks, UC Berkeley professor of integrative biology, but endurance training teaches the body to efficiently use lactic acid as a source of fuel on par with the carbohydrates stored in muscle tissue and the sugar in blood. Efficient use of lactic acid, or lactate, not only prevents lactate build-up, but ekes out more energy from the body's fuel.In a paper in press for the American Journal of Physiology - Endocrinology and Metabolism, published online in January, Brooks and colleagues Takeshi Hashimoto and Rajaa Hussien in UC Berkeley's Exercise Physiology Laboratory add one of the last puzzle pieces to the lactate story and also link for the first time two metabolic cycles - oxygen-based aerobic metabolism and oxygen-free anaerobic metabolism - previously thought distinct.
"This is a fundamental change in how people think about metabolism," Brooks said. "This shows us how lactate is the link between oxidative and glycolytic, or anaerobic, metabolism."
He and his UC Berkeley colleagues found that muscle cells use carbohydrates anaerobically for energy, producing lactate as a byproduct, but then burn the lactate with oxygen to create far more energy. The first process, called the glycolytic pathway, dominates during normal exertion, and the lactate seeps out of the muscle cells into the blood to be used elsewhere. During intense exercise, however, the second ramps up to oxidatively remove the rapidly accumulating lactate and create more energy.
Training helps people get rid of the lactic acid before it can build to the point where it causes muscle fatigue, and at the cellular level, Brooks said, training means growing the mitochondria in muscle cells. The mitochondria - often called the powerhouse of the cell - is where lactate is burned for energy.
"The world's best athletes stay competitive by interval training," Brooks said, referring to repeated short, but intense, bouts of exercise. "The intense exercise generates big lactate loads, and the body adapts by building up mitochondria to clear lactic acid quickly. If you use it up, it doesn't accumulate."
To move, muscles need energy in the form of ATP, adenosine triphosphate. Most people think glucose, a sugar, supplies this energy, but during intense exercise, it's too little and too slow as an energy source, forcing muscles to rely on glycogen, a carbohydrate stored inside muscle cells. For both fuels, the basic chemical reactions producing ATP and generating lactate comprise the glycolytic pathway, often called anaerobic metabolism because no oxygen is needed. This pathway was thought to be separate from the oxygen-based oxidative pathway, sometimes called aerobic metabolism, used to burn lactate and other fuels in the body's tissues.
Experiments with dead frogs in the 1920s seemed to show that lactate build-up eventually causes muscles to stop working. But Brooks in the 1980s and '90s showed that in living, breathing animals, the lactate moves out of muscle cells into the blood and travels to various organs, including the liver, where it is burned with oxygen to make ATP. The heart even prefers lactate as a fuel, Brooks found.
Brooks always suspected, however, that the muscle cell itself could reuse lactate, and in experiments over the past 10 years he found evidence that lactate is burned inside the mitochondria, an interconnected network of tubes, like a plumbing system, that reaches throughout the cell cytoplasm.
In 1999, for example, he showed that endurance training reduces blood levels of lactate, even while cells continue to produce the same amount of lactate. This implied that, somehow, cells adapt during training to put out less waste product. He postulated an "intracellular lactate shuttle" that transports lactate from the cytoplasm, where lactate is produced, through the mitochondrial membrane into the interior of the mitochondria, where lactate is burned. In 2000, he showed that endurance training increased the number of lactate transporter molecules in mitochondria, evidently to speed uptake of lactate from the cytoplasm into the mitochondria for burning.
The new paper and a second paper to appear soon finally provide direct evidence for the hypothesized connection between the transporter molecules - the lactate shuttle - and the enzymes that burn lactate. In fact, the cellular mitochondrial network, or reticulum, has a complex of proteins that allow the uptake and oxidation, or burning, of lactic acid.
"This experiment is the clincher, proving that lactate is the link between glycolytic metabolism, which breaks down carbohydrates, and oxidative metabolism, which uses oxygen to break down various fuels," Brooks said.
Post-doctoral researcher Takeshi Hashimoto and staff research associate Rajaa Hussien established this by labeling and showing colocalization of three critical pieces of the lactate pathway: the lactate transporter protein; the enzyme lactate dehydrogenase, which catalyzes the first step in the conversion of lactate into energy; and mitochondrial cytochrome oxidase, the protein complex where oxygen is used. Peering at skeletal muscle cells through a confocal microscope, the two scientists saw these proteins sitting together inside the mitochondria, attached to the mitochondrial membrane, proving that the "intracellular lactate shuttle" is directly connected to the enzymes in the mitochondria that burn lactate with oxygen.
"Our findings can help athletes and trainers design training regimens and also avoid overtraining, which can kill muscle cells," Brooks said.